[i]Originally posted by JJ[/i]
I call it neuromuscular speed endurance b/c I contend (with absolutely no proof) that this protocol (and similar ones) may help the athlete be able to modify the refractory state of the Na+/K+ pump at the neuromuscular junction. Why is this important? I have a suspicion that this may be one of the mechanisms of fatigue in the 100m dash. Personally, I don't believe that lactate plays a part in the deceleration phase of the 100m. I do, however, believe that the glycolytic system plays a role, but that is another post for another time.
Two different possible mech's of fatigue:
1) I'm speaking of what is happening at the level of the Na+/K+ pump that creates the action potential presynaptically.
2) The sarcoplasmic reticulum (which the T-tubules are a part of) could also be a site for fatigue at the postsynaptic level.
JJ, could you elaborate more on this? Are you saying that the body can't supply enough Na+/K+ to keep up with the demand and therefore deceleration occurs?